CALL FOR PAPERS Biomarkers in Lung Diseases: from Pathogenesis to Prediction to New Therapies IL-6 trans-signaling increases expression of airways disease genes in airway smooth muscle

نویسندگان

  • Mac B. Robinson
  • Deepak A. Deshpande
  • Jeffery Chou
  • Wei Cui
  • Shelly Smith
  • Carl Langefeld
  • Annette T. Hastie
  • Eugene R. Bleecker
  • Gregory A. Hawkins
چکیده

Mac B. Robinson, Deepak A. Deshpande, Jeffery Chou, Wei Cui, Shelly Smith, Carl Langefeld, Annette T. Hastie, Eugene R. Bleecker, and Gregory A. Hawkins Wake Forest School of Medicine, Center for Genomics and Personalized Medicine Research, Winston-Salem, North Carolina; Wake Forest School of Medicine, Department of Neurobiology and Anatomy, Winston-Salem, North Carolina; Center for Translational Medicine, Thomas Jefferson University, Philadelphia, Pennsylvania; and Wake Forest School of Medicine, Center for Public Health Genomics, Winston-Salem, North Carolina

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

IL-6 trans-signaling increases expression of airways disease genes in airway smooth muscle.

Genetic data suggest that IL-6 trans-signaling may have a pathogenic role in the lung; however, the effects of IL-6 trans-signaling on lung effector cells have not been investigated. In this study, human airway smooth muscle (HASM) cells were treated with IL-6 (classical) or IL-6+sIL6R (trans-signaling) for 24 h and gene expression was measured by RNAseq. Intracellular signaling and transcripti...

متن کامل

Differential genes expression analysis of invasive aspergillosis: a bioinformatics study based on mRNA/microRNA

Invasive aspergillosis is a severe opportunistic infection with high mortality in immunocompromised patients. Recently, the roles of microRNAs have been taken into consideration in the immune system and inflammatory responses. Using bioinformatics approaches, we aimed to study the microRNAs related to invasive aspergillosis to understand the molecular pathways involved in the disease pathogenes...

متن کامل

CALL FOR PAPERS Biomarkers in Lung Diseases: from Pathogenesis to Prediction to New Therapies Resolvin E1 normalizes contractility, Ca sensitivity and smooth muscle cell migration rate in TNF- - and IL-6-pretreated human pulmonary arteries

Roddy Hiram, Edmond Rizcallah, Sofia Marouan, Chantal Sirois, Marco Sirois, Caroline Morin, Samuel Fortin, and Eric Rousseau Department of Pharmacology and Physiology, Université de Sherbrooke, Sherbrooke, Quebec, Canada; Department of Obstetrics-Gynecology, Université de Sherbrooke, Sherbrooke, Quebec, Canada; Department of Pathology, Université de Sherbrooke, Sherbrooke, Quebec, Canada; Servi...

متن کامل

IL6 Trans-signaling Increases Expression of Airways Disease Genes in Airway Smooth

23 Genetic data suggests that IL6 trans-signaling may have a pathogenic role in the lung; however, 24 the effects of IL6 trans-signaling on lung effector cells have not been investigated. In this study, 25 human airway smooth muscle (HASM) cells were treated with IL6 (classical) or IL6+sIL6R 26 (trans-signaling) for 24 h and gene expression measured by RNAseq. Intracellular signaling and 27 tra...

متن کامل

CALL FOR PAPERS Biomarkers in Lung Diseases: from Pathogenesis to Prediction to New Therapies Novel identification of the free fatty acid receptor FFAR1 that promotes contraction in airway smooth muscle

Kentaro Mizuta, Yi Zhang, Fumiko Mizuta, Hiroshi Hoshijima, Toshiya Shiga, Eiji Masaki, and Charles W. Emala Sr. Department of Anesthesiology, College of Physicians and Surgeons of Columbia University, New York, New York; Department of Dento-oral Anesthesiology, Tohoku University Graduate School of Dentistry, Sendai, Japan; and Department of Anesthesiology, Chemotherapy Research Institute, Kake...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2015